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  • Benzodiazepine Pharmacology and Central Nervous System–Mediated Effects
    BZDs act as positive allosteric modulators on the gamma amino butyric acid (GABA)-A receptor The GABA-A receptor is a ligand-gated chloride-selective ion channel GABA is the most common neurotransmitter in the central nervous system, found in high concentrations in the cortex and limbic system
  • GABA in Psychology: Neurotransmitters Mental Health Impact
    GABA, or gamma-aminobutyric acid, is the primary inhibitory neurotransmitter in the central nervous system In simpler terms, it’s like the brain’s brake pedal, slowing down neural activity and preventing overstimulation But how does this tiny molecule exert such a powerful influence?
  • Understanding GABA: The Neurotransmitter of Calm and its Role in Mental . . .
    Gamma-Aminobutyric Acid (GABA) is a crucial neurotransmitter in the brain that plays a significant role in regulating anxiety, stress, and overall mental health This comprehensive blog post explores the science behind GABA, its impact on mental health, and practical strategies to enhance its effects for better well-being What is GABA?
  • Peripheral GABA A receptors - Physiological relevance and . . . - PubMed
    GABA A receptors located outside the CNS ("peripheral GABA A receptors") enable both neuronal and non-neuronal GABA-ergic signaling in various physiological processes and are generally thought to have similar properties to the extrasynaptic receptors in the CNS
  • The role of GABA neurotransmitter in the human central nervous system . . .
    In depression and anxiety, studies indicate changes in the regulation of the genes which encoding GABA receptors In the same way, GABA receptors have been associated with alcoholism and
  • GABA and the blood-brain barrier – Troscriptions®
    GABAergic signaling is involved in several psychiatric and neurological disorders such as anxiety, depression, epilepsy, and schizophrenia [3] Deficit in GABA can lead to insomnia, irritability, attention deficit hyperactivity disorder, or epilepsy This is why the oral ingestion of GABA is done today as a food supplement
  • The GABAergic System: An Overview of Physiology, Physiopathology and . . .
    Gamma-aminobutyric acid (GABA) is the main inhibitory neurotransmitter in the central nervous system, where it is widely distributed GABA has an important role in neurodevelopment, and depending on the period of development, its action can be excitatory or inhibitory
  • GABA receptor-mediated effects in the peripheral nervous system: A . . .
    Gamma-aminobutyric acid (GABA), the major inhibitory neurotransmitter in the adult mammalian central nervous system (CNS), exerts its action via an interaction with specific receptors (e g , GABAA and GABAB)
  • The Roles of GABA in Ischemia‐Reperfusion Injury in the Central Nervous . . .
    It is now known that GABA primarily plays inhibitory effects on neuronal excitabilities of postsynaptic membrane in the central nervous system of adults [19] GABA can also relax muscle at neuromuscular synapses [20]
  • Peripheral GABA - PubMed
    Unlike in the central nervous system (CNS), in the adult peripheral nervous system (PNS), activation of GABA A receptors (GABA A R) is excitatory because of the relatively high concentration of intracellular chloride in these neurons
  • GABA receptor-mediated effects in the peripheral nervous system - Springer
    γ-Aminobutyric acid (GABA), the major inhibitory neurotransmitter in the adult mammalian central nervous system (CNS), exerts its action via an interaction with specific receptors (e g , GABA A and GABA B)
  • Peripheral GABA - ScienceDirect
    GABA A receptors located outside the CNS (“peripheral GABA A receptors”) enable both neuronal and non-neuronal GABA-ergic signaling in various physiological processes and are generally thought to have similar properties to the extrasynaptic receptors in the CNS
  • Stress-induced blood brain barrier disruption: Molecular . . . - PubMed
    In this review, we summarize studies conducted on the effects of stress on the BBB and integrate recent data that suggest possible molecular mechanisms and signaling pathways underlying stress-induced BBB disruption Key molecular targets and pharmacological candidates for treatment of stress and related illnesses are also summarized





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