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  • Thyroid hormone action and liver disease, a complex interplay
    TH is also involved in the reverse cholesterol transport pathway increasing expression of CYP7A1 Furthermore, TH increases the efflux of bile by stimulating Abcg5 Abcg8 gene transcription Additionally, TH can negatively modulate cholesterol ester formation by means of CDX2 and SOAT2
  • IL-1 and EGF regulate expression of genes important in inflammation and . . .
    This review focuses on the mechanisms by which the expression of specific genes is regulated by two proteins that are important in inflammation and cancer, namely the pro-inflammatory cytokine interleukin (IL)-1β and epidermal growth factor (EGF)
  • Dysregulation of Inflammasome Priming and Activation by . . . - PubMed
    Priming and activation of inflammasomes can be modulated by microRNAs (miRNAs), small noncoding RNAs that regulate gene expression posttranscriptionally miRNAs, such as miR-223-3p, have been demonstrated to directly target the inflammasome components NLRP3, caspase-1, and caspase-8
  • The Pro-Inflammatory Cytokine Cascade | SpringerLink
    Different experimental approaches have demonstrated the contribution of the pro-inflammatory cytokines to the harmful effects observed in sepsis, trauma, burns, hemorrhage, severe surgery and other pathophysiological situations leading to systemic inflammatory response syndrome (SIRS)
  • Regulation of Inflammatory Reaction in Health and Disease
    The activation of transcription and translation of specific pro-inflammatory genes is manifested by the release of the cytokines in question, IL-1, TNF-α, and IL-6 At the same time, the oxidative damage produced by the inflammatory trigger reduces the production of AMP-kinase (AMPK), a fundamental inhibitory control factor of NF-kB translocation
  • Chapter 1 - Macrophage Proinflammatory Activation and Deactivation : A . . .
    LPS recognition requires the binding of LPS to lipoprotein binding protein (LBP) and CD14 in the surface of macrophages Signal transduction to LPS is then triggered by toll-like receptor 4 (TLR-4), which results in coordinate transcriptional activation of proinflammatory gene expression
  • Proinflammatory cytokines, aging, and age-related diseases
    The low-grade inflammation characterizing the aging process notably concurs at the pathophysiological mechanisms underlying sarcopenia In addition, proinflammatory cytokines (through a variety of mechanisms, such as platelet activation and endothelial activation) may play a major role in the risk of cardiovascular events
  • Proinflammatory Cytokines - CHEST
    What triggers the expression of these genes? Although inflammatory products such as endotoxins trigger it, the cytokines IL-1 and TNF (and in some cases IFN-γ) are particularly effective in stimulating the expression of these genes Moreover, IL-1 and TNF act synergistically in this process
  • Frontiers | Approaching Inflammation Paradoxes—Proinflammatory Cytokine . . .
    Our findings have provided novel insights on various inflammation paradoxes and proinflammatory cytokines regulation of innate immune regulators; and may re-shape new therapeutic strategies for cardiovascular disease and other inflammatory diseases
  • Cytokines in Inflammatory Disease - PMC
    Furthermore, it leads to the induction, production and the release of pro-inflammatory mediators, including cytokines such as tumor necrosis factor-alpha (TNF-α), interleukin (IL)-1, IL-6 and IL-10, or type 1 interferons (IFNs) [15]





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