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  • Ovarian low-grade and high-grade serous carcinoma: pathogenesis . . .
    Low-grade serous carcinomas exhibit low-grade nuclei with infrequent mitotic figures They evolve from adenofibromas or borderline tumors, have frequent mutations of the KRAS, BRAF, or ERBB2 genes, and lack TP53 mutations (Type I pathway) The progression to invasive carcinoma is a slow step-wise process
  • OVARIAN LOW-GRADE AND HIGH-GRADE SEROUS CARCINOMA: Pathogenesis . . .
    Low-grade serous carcinomas exhibit low-grade nuclei with infrequent mitotic figures They evolve from adenofibromas or borderline tumors, have frequent mutations of the KRAS, BRAF, or ERBB2 genes, and lack TP53 mutations (Type I pathway) The progression to invasive carcinoma is a slow step-wise process
  • Origin and molecular pathogenesis of ovarian high-grade serous carcinoma
    Treatment of low-grade and high-grade serous carcinoma, like early detection, should be different based on their distinctive molecular pathways Low-grade serous tumors are slow growing and are confined to the ovary at diagnosis usually spreading late in their evolution
  • Ovarian low-grade and high-grade serous carcinoma: pathogenesis . . .
    Low-grade serous carcinomas exhibit low-grade nuclei with infrequent mitotic figures They evolve from adenofibromas or borderline tumors, have frequent mutations of the KRAS, BRAF, or ERBB2 genes, and lack TP53 mutations (Type I pathway) The progression to invasive carcinoma is a slow step-wise process
  • Ovarian Low-grade and High-grade Serous Carcinoma - LWW
    Ovarian serous carcinomas have been graded using various systems Recently, a 2-tier system in which tumors are subdivided into low grade and high grade has been proposed This approach is simplistic, reproducible, and based on biologic evidence indicating that both tumors develop via different pathways
  • Fallopian tube precursors of ovarian low‐ and high‐grade serous . . .
    This review paper provides an overview of the fallopian tube's potential role in the pathogenesis of both types of serous neoplasms of the ovary Traditionally, it was thought that ovarian high-grade serous carcinoma arises from the ovarian surface epithelium and epithelial inclusion glands and that the pathogenesis is de novo; nonetheless,
  • Clinicopathologic Features, Molecular Landscape, and Prognostic . . .
    Severe nuclear atypia and absence of RAS RAF-driver mutations were significant adverse prognostic factors Conclusions: LGS-HT exhibit both low-grade and high-grade morphologic and molecular features, representing an exception to the dualistic classification of ovarian serous neoplasms
  • Overcoming intra-tumoral heterogeneity for biomarker discovery in the . . .
    Improved biomarkers of treatment response are needed for patients with high-grade serous ovarian cancer (HGSC) A challenge is substantial anatomical site-to-site variation in expression
  • Ovarian low and high grade serous carcinomas: hidden divergent features . . .
    Only recently low-grade serous carcinoma (LGSOC) of the ovary has been recognized as a disease entity distinct from the more common high-grade serous carcinoma (HGSOC), with significant differences in pathogenesis and clinical and pathologic
  • Ovarian low-grade and high-grade serous carcinoma . . .
    Low-grade serous carcinomas exhibit low-grade nuclei with infrequent mitotic figures They evolve from adenofibromas or borderline tumors, have frequent mutations of the KRAS, BRAF, or ERBB2 genes, and lack TP53 mutations (Type I pathway) The progression to invasive carcinoma is a slow step-wise process





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