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  • Testosterone-induced suppression of lipoprotein(a) in normal men . . .
    Two studies [12,13] using oral andro- gen therapy of osteoporosis or endometriosis in women have reported a suppressive effect on Lp(a) levels However, interpretation of this work is difficult because oral androgens exert larger effects on the liver (because of high concentrations in the portal circulation) than the parenterally administered
  • Testosterone-induced suppression of lipoprotein(a) in normal men . . .
    The concentration of lipoprotein(a) [Lp(a)] in human plasma is largely genetically determined and is inversely correlated to the size of apolipoprotein(a) [apo(a)] Additionally, Lp(a) values are relatively stable within individuals and are only marginally susceptible to therapeutic treatment The aim of our study was to evaluate the effect of exogenous testosterone on plasma Lp(a) concentration
  • Testosterone decreases lipoprotein (a) in men - ScienceDirect
    Testosterone is normally aromatized to estradiol, and peripheral aromatization of testosterone is the major source of circulating estrogen in men 5 We recently examined the effect of testosterone aromatization on serum lipid and lipoprotein levels in men by administering testosterone alone or in combination with the aromatase inhibitor
  • Testosterone decreases lipoprotein(a) in men - American Journal of . . .
    In summary, the results of the present study indicate that testosterone reduces Lp(a) concentrations in normal men primarily by an androgenic effect and not by its conversion to estradiol Testosterone decreases lipoprotein(a) in men - American Journal of Cardiology
  • Effect of testosterone replacement therapy on lipids and lipoproteins . . .
    We investigated the effects of long-term testosterone replacement in hypogonadal and elderly men on lipids and lipoproteins Twenty-two men with initial serum testosterone concentrations below 3 5 ng ml took part in the study: 11 with hypopituitarism (1st group) and 11 otherwise healthy elderly men with low testosterone levels (2nd group)
  • Suppression of Endogenous Testosterone in Young Men Increases Serum . . .
    Antagonists of the GnRH suppress testosterone levels and cause increases in HDL cholesterol dependent on dosage and time (16– 18) In this study we investigated the effect of testosterone suppression on lipid metabolism in healthy young men who received the GnRH antagonist Cetrorelix (19, 20) over a period of 3 weeks
  • The benefit and risk of testosterone replacement therapy in older men . . .
    Effect of testosterone replacement therapy on serum ApoA1, ApoB and lipoprotein(a) (Lp[a]) plasma levels (17) On the whole the results of these particular stud- crease in HDL-cholesterol was lower in the studies Table 1 Effect of testosterone replacement therapy on lipid fractions: results of published meta-analyses
  • Physiological Testosterone Replacement Therapy Attenuates Fatty Streak . . .
    ing levels may lead to a proatherosclerotic environment (reviewed elsewhere3) Clinical Perspective p 2434 Beneficial effects of androgen supplementation in athero-genesis have been described in 4 animal studies Testosterone replacement has been demonstrated to prevent aortic choles-terol accumulation in cholesterol-fed, orchidectomized male
  • Risks of Testosterone-Replacement Therapy and Recommendations for . . .
    between higher testosterone levels and heart dis-ease 23-26 Indeed, several studies suggest that high-er testosterone levels may actually have a favorable effect on the risk of cardiovascular
  • Can Testosterone Affect Your Cholesterol Levels? Find Out From The . . .
    A 2021 study found that low testosterone levels can increase the risk of cardiovascular problems, while testosterone replacement therapy, which boosts those levels, may reduce the likelihood of
  • Effect of transdermal testosterone treatment on serum lipid and . . .
    Levels of high-density lipoprotein (HDL) cholesterol, triglycerides, and apolipoproteins A-I and B did not change Lipoprotein(a) levels increased in both groups by similar amounts (testosterone treated, 3 ± 9 mg dL; placebo treated, 4 ± 6 mg dL; P = 1 0) The number of cardiovascular events was small and did not differ significantly between





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